Post by LymeEnigma on Jun 14, 2008 10:36:09 GMT -8
Antibiotic induced bacterial killing activates vascular endothelial cells and whole blood cells
Journal of Endotoxin Research, Vol. 3, No. 3, 179-185 (1996)
DOI: 10.1177/096805199600300303
© 1996 SAGE Publications
The results support the hypothesis that free endotoxin released from E. coli following bacteriolysis is the major contributor to the enhancement of the inflammation associated with the initiation of antibiotic therapy. The dichotomy of IL-10 effects on LPS-induced cytokine production in blood mononuclear cells versus endothelial cells may play a role in the pathophysiology of TH-2-directed responses.
Abstract: ini.sagepub.com/cgi/content/abstract/3/3/179
Journal of Endotoxin Research, Vol. 3, No. 3, 179-185 (1996)
DOI: 10.1177/096805199600300303
© 1996 SAGE Publications
The results support the hypothesis that free endotoxin released from E. coli following bacteriolysis is the major contributor to the enhancement of the inflammation associated with the initiation of antibiotic therapy. The dichotomy of IL-10 effects on LPS-induced cytokine production in blood mononuclear cells versus endothelial cells may play a role in the pathophysiology of TH-2-directed responses.
Abstract: ini.sagepub.com/cgi/content/abstract/3/3/179